Class: Polyclonal T-cell–depleting immunoglobulin
Indication: Prophylaxis and treatment of acute rejection in kidney transplantation (U.S. label), widely used off-label for induction in high immunologic risk patients and in steroid-resistant rejection.


Mechanism of Action

Thymoglobulin is a polyclonal IgG antibody preparation derived from rabbits immunized with human thymocytes. It contains antibodies against multiple T-cell surface antigens (CD2, CD3, CD4, CD8, CD11a, CD18, HLA-DR, CD28, β2-microglobulin), resulting in:

  • Complement-mediated T-cell lysis
  • Opsonization-induced phagocytosis
  • Apoptosis of T cells
  • Modulation of adhesion and trafficking molecules
  • Upregulation of T-regulatory cells & immunomodulation

Profound T-cell depletion occurs within 24 hours and may persist weeks to months.


Clinical Use Overview

SettingTypical DoseDurationNotes
Induction in kidney transplant1–1.5 mg/kg/day IV3–7 daysAdminister before graft reperfusion
Treatment of acute rejection1.5 mg/kg/day IV7–14 daysSteroid-resistant cellular rejection
Off-label (U.S. centers)Aplastic anemia, GVHD prophylaxisVariableHematology/oncology protocols

Dosing per U.S. label: 1.5 mg/kg/day x 4–7 days (prophylaxis) or 7–14 days (treatment)

Antithymocyte globulin (ATG) induction

Antithymocyte globulin (ATG) induction refers to the use of T-cell–depleting immunoglobulin therapy—most commonly Thymoglobulin (rabbit ATG)—administered at the time of organ transplantation to profoundly suppress the recipient’s immune system and prevent early acute rejection.

Purpose of ATG Induction
ATG induction therapy is designed to achieve rapid, profound, but temporary depletion of circulating T lymphocytes during the peri-transplant period, when alloimmune activation against the new organ is most intense.

  • It provides immediate immunosuppression before maintenance drugs (calcineurin inhibitors, mycophenolate, prednisone) can reach full effect.
  • It is used both in standard immunologic risk and especially in high immunologic risk recipients (e.g., repeat transplant, sensitized patients, African American recipients, presence of donor-specific antibodies).

Preparation Guide

Reconstitution

  • Each vial: 25 mg lyophilized powder.
  • Reconstitute with 5 mL Sterile Water for Injection (SWFI).
  • Concentration: 5 mg/mL.
  • Gently invert—do NOT shake.

Dilution

  • Dilute dose in 0.9% NaCl or D5W to 50–500 mL total volume.
  • Use 0.22 micron filter inline.
  • Infusion time:
    • First dose ≥ 6 hours, subsequent doses ≥ 4 hours.

Peripheral IV Administration Precaution

  • If given peripherally, MUST add to infusion:
    • Heparin 1,000 units + Hydrocortisone 50 mg in 0.9% NaCl (not D5W – precipitation risk)
  • Use central line if possible.

Beyond Use Date (BUD)

Preparation Stage Storage Condition Maximum BUD Key Comments
Reconstituted vial (not yet diluted) 2–8 °C 24 h Protect from light; single-use only
Diluted in 0.9% NaCl IV solution Room temperature or 2–8 °C 24 h Includes infusion time; discard remainder
Compatibility/Incompatibility Data

Thymoglobulin is a highly proteinaceous biologic sensitive to pH, ionic strength, and the carbohydrate content of the diluent. Proper diluent selection is critical for preventing precipitation and maintaining product integrity during infusion.


Preferred Diluent and Compatible Solutions

  • Compatible Diluent: 0.9% Sodium Chloride Injection (Normal Saline) – the only manufacturer-recommended and validated diluent.
  • Final Concentration: Do not exceed 0.5 mg/mL after dilution in 0.9% NaCl.
  • Rationale: Saline maintains neutral pH and isotonicity, preserving protein structure and preventing aggregation.

Incompatibility with Dextrose-Containing Solutions (D5W)

  • Thymoglobulin is incompatible with dextrose-containing solutions (e.g. D5W).
  • Precipitation occurs, especially when mixed with heparin and hydrocortisone.
  • Mechanism: Carbohydrate interaction and loss of ionic balance disrupt electrostatic stability, causing protein aggregation.
  • Practice Rule: Do not substitute D5W as a diluent or co-infuse with Thymoglobulin.

Co-Administration and IV Line Use

  • Y-site compatibility: No supporting data; manufacturer advises against mixing with other drugs in the same line.
  • Best Practice: Use a dedicated IV line for Thymoglobulin, or flush thoroughly with saline before and after if shared access is unavoidable.
  • Filter Requirement: Administer through a 0.22 µm in-line filter to remove microaggregates.

Physical and Chemical Stability

Diluent Compatibility Precipitation Risk Notes
0.9% Sodium Chloride Compatible Low Preferred diluent; maintains protein stability
D5W (5% Dextrose) Incompatible High Causes precipitation, especially with heparin/hydrocortisone
Ringer’s Lactate Not evaluated Unknown Possible calcium–protein interaction risk
Plasma-Lyte or mixed electrolytes Limited data Not recommended Ionic imbalance may destabilize biologic

Clinical and Safety Implications

Saline is the clinically preferred diluent due to its stability and compatibility with biologic proteins. Dextrose-based solutions increase precipitation risk, reduce potency, and may lead to embolic risk from protein aggregates.


Key Professional Takeaways

  • Always dilute Thymoglobulin in 0.9% NaCl only.
  • Do not use D5W – high precipitation and instability risk.
  • Use a 0.22 µm in-line filter for all infusions.
  • Do not mix with other drugs in the same IV line.
  • Use within 24 hours of compounding (even if refrigerated).

Premedication

30–60 min prior to each dose:

MedicationRationale
AcetaminophenPrevent fever/chills
DiphenhydramineReduce histamine-mediated reactions
MethylprednisolonePrevent cytokine release syndrome (CRS)

Supportive Therapy

Prophylaxis

ClassAgent Examples
AntiviralValganciclovir (CMV risk)
AntifungalFluconazole
PJP prophylaxisTMP–SMX

Infusion Reactions & CRS

High-risk during first 1–2 doses due to cytokine release.
Symptoms: fever, chills, hypotension, dyspnea, tachycardia, ARDS, chest pain

Management:

  • Slow infusion rate.
  • Additional steroids + antihistamines PRN.
  • For anaphylaxis: stop infusion + epinephrine 0.3–0.5 mL SC (1:1000).

Adverse Effects

Adverse Effect Incidence
Fever / chills 46–63%
Leukopenia 57–63%
Thrombocytopenia 10–36%
Hypertension 18–37%
Infection (overall) 56–76%
CMV infection Up to 12–18%
Serum sickness (day 5–15) 1–5%
Malignancy risk PTLD cases reported
Anaphylaxis Rare but fatal cases reported

Monitoring Parameters

ParameterFrequency
CBC with diff, plateletsDaily
Vital signs during infusionq15–30 min
CMP, K⁺, Mg²⁺Daily
CMV PCR, EBV loadWeekly (if high risk)
T-cell counts (CD3)Optional for redosing
Signs of serum sicknessDay 5–15

Hold or reduce dose if:

  • WBC 2,000–3,000 → give ½ dose
  • WBC <2,000 or Plt <50,000HOLD therapy

Professional Clinical Pearls

  • Avoid mixing in dextrose if giving heparin/hydrocortisone
  • Start infusion BEFORE reperfusion to reduce delayed graft function
  • Consider lower steroid exposure protocols with ATG induction
  • Not effective for antibody-mediated rejection (AMR)—use plasmapheresis + IVIG
  • Serum sickness risk increases with retreatment—watch for fever/arthralgia
  • Use CMV prophylaxis in D+/R− transplants
  • Thymoglobulin provides broad and powerful immunosuppression but requires meticulous infection control and hematologic monitoring.
  • Dosing must be product-specific, as potency differs among ATG formulations (rabbit vs equine).
  • Premedication, slow infusion, and vigilant supportive care are essential to minimize adverse events and optimize graft outcomes.

References

  1. U.S. Food and Drug Administration. Thymoglobulin (Anti-thymocyte Globulin [Rabbit]) Package Insert. Revised 2024. Accessed October 26, 2025. https://www.fda.gov/media/74641/download
  2. Sanofi-Aventis Canada Inc. Thymoglobulin (Anti-thymocyte Globulin [Rabbit]) Product Monograph. Toronto, ON: Sanofi-Aventis Canada Inc.; Revised March 7, 2016. Accessed October 26, 2025. https://www.sanofi.com/assets/countries/canada/docs/products/prescription-products/thymoglobulin-en.pdf
  3. Noël C, Abramowicz D, Durand D, et al. Daclizumab versus antithymocyte globulin in high-immunological-risk renal transplant recipients. Nephrol Dial Transplant. 2017;32(10):1601-1609. https://academic.oup.com/ndt/article/32/10/1601/2374143
  4. Gaber AO, Knight RJ, Patel SJ, et al. Rabbit antithymocyte globulin induction versus no induction in renal transplantation: three-year follow-up of the Thymoglobulin Induction Study Group trial. PLoS One. 2016;11(1):e0146238. https://pmc.ncbi.nlm.nih.gov/articles/PMC4689936/
  5. Sanofi Genzyme. Thymoglobulin: Efficacy and Safety Data. Accessed October 26, 2025. https://www.thymoglobulin.com/efficacy-safety-data
  6. Brennan DC, Daller JA, Lake KD, Cibrik D, Del Castillo D. Rabbit antithymocyte globulin versus basiliximab in renal transplantation. N Engl J Med. 2006;355(19):1967-1977. https://pubmed.ncbi.nlm.nih.gov/28376289/
  7. Sanofi Genzyme. Thymoglobulin: Dosing and Administration Guide. Accessed October 26, 2025. https://www.thymoglobulin.com/dosing